By Arief A Suriawinata MD, Swan N. Thung MD
Liver Pathology: An Atlas and Concise consultant offers pathologists, hepatologists, gastroenterologists, citizens and fellows of their respective fields with an updated advisor to the differential prognosis, interpretation and analysis of liver specimens. The ebook publications the reader to appreciate universal histologic styles and key pathologic beneficial properties of the extra universal liver problems. Liver Pathology: An Atlas and Concise advisor comprises over six hundred high quality colour photos demonstrating the histopathologic and immunohistichemical findings supported by means of concise textual content together with usually linked medical findings, pathologic gains (histology, immunohistochemistry, molecular studies), differential diagnoses, and key references. Liver Pathology: An Atlas and Concise consultant presents the "fundamentals" of liver pathology in a simple, problem-oriented presentation of key issues and common differentials in accordance with what you are going to see. Liver Pathology: An Atlas and Concise advisor comprises concentrate on vintage positive factors that each pathologist is probably going to determine Differential diagnoses offered in tables for effective evaluate enormous quantities of top quality photos chosen to illustrate key pathologic beneficial properties and emphasize differential prognosis chosen references and evaluate articles to guide the reader to extra research while wanted
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Additional resources for Liver Pathology: An Atlas and Concise Guide
In patients with Mycobacterium aviumintracellulare, clusters of macrophages are seen filled with the organisms that are positive for periodic acid–Schiff with diastase digestion. Drug-induced injury may result in granuloma formation and may be accompanied by acute hepatitis or cholestatic hepatitis. 4). The more common drugs that cause granuloma are allopurinol, sulfonamides, phenytoin, carbamazepine, quinine, thiabendazole, and phenylbutazone. Hodgkin and non-Hodgkin lymphomas can cause nonspecific granulomatous reaction in the liver.
1 Hepatocytes are swollen with pale and rareﬁed cytoplasm in ballooning degeneration. 2 Acidophilic body (arrow) signifying apoptotic hepatocyte, found in an otherwise normal liver or in abundance in acute hepatocellular injury. 3 Mitosis signifying hepatocellular regeneration. It is rarely encountered in an otherwise normal liver as a physiological turnover. 4 The PAS reaction after diastase demonstrates clusters of macrophages, engulﬁng hepatocellular debris after an episode of hepatocellular injury.
The prognosis of nodular regenerative hyperplasia is determined by its complications, particularly portal hypertension. Hepatoportal Sclerosis Hepatoportal sclerosis, also known as idiopathic portal hypertension, noncirrhotic portal hypertension, or obliterative portal venopathy, denotes a condition consisting of portal hypertension, splenomegaly, and anemia secondary to hypersplenism in a noncirrhotic patient with dense portal tract fibrosis and obliteration of portal veins. One of the known causes of hepatoportal sclerosis is chronic exposure to copper, arsenic, and vinyl chloride.